Allergic eye disease
Mnemonic: HFS
H Delayed Hypersensitivity
F HayFever conjunctivitis
S Spring catarrh
Clinical presentation of conjunctivitis
Mnemonic: BURN
B Burning and lacrimation along with itching and possibly photophobia
U Usually bilateral, if unilateral consider another differential diagnosis
R Red and inflamed conjunctiva, eyelids may be stuck together with purulent
discharge
N Normally self-limiting, can be treated with antibiotics
Signs of optic neuropathy
Mnemonic: PLAC
P Pale disc
L Loss of visual acuity/Loss of red colour vision
A Afferent pupillary defect
C Central scotoma
Causes of retinal detachment
Mnemonic: SITS
S Secondary to some intraocular problem (melanoma)
I Idiopathic
T Trauma
S Surgery for cataract
The 4 Fs of retinal detachment
Mnemonic: 4 Fs
F Floaters (small dark spots on a bright background are generally harmless)
F Flashes (migraine)
F Field loss (dark cloud covers a field of vision)
F Falling acuity (serious)
Causes of excess lacrimation
Mnemonic: FACE
F Foreign body or corneal abrasions
A Acute glaucoma
C Conjunctivitis
E Emotion (typical man, I list this last!)
Remember Keratoconus with the help of CONES:
Central scarring & Fleischer ring
Oil drop reflex / Oedema (hydrops)
Nerves prominent
Excessive bulging of lower lid on downgaze (Munson’s sign)
Striae (Vogt’s)
Remember Iridocorneal Endothelial Syndrome with ICE
Iris Naevus
Chandler Syndrome
Essential Iris Atrophy
Remember Behcet's Disease with the help of ORAL UPSET
Occlusive periphlebitis
Retinitis
Anterior uveitis
Leakage from retinal vessels
Ulceration (aphthous/genital)
Pustules after skin trauma (Pathergy test)
Scratching leaves lines (dermatographism)
Erythema nodosum
Thrombophlebitis
Remember posterior scleritis with POST SCLER
Proptosis
Ophthalmoplegia
Swelling of disc
Thickening of sclera (US/CT) & T sign (fluid in sub-Tenon’s space)
Subretinal exudates
Choroidal foLds
Exudative RD
Ring choroidal detachment
Remember Keratoconus with the help of CONES:
Central scarring & Fleischer ring
Oil drop reflex / Oedema (hydrops)
Nerves prominent
Excessive bulging of lower lid on downgaze (Munson’s sign)
Striae (Vogt’s)
Remember Iridocorneal Endothelial Syndrome with ICE
Iris Naevus
Chandler Syndrome
Essential Iris Atrophy
Remember Behcet's Disease with the help of ORAL UPSET
Occlusive periphlebitis
Retinitis
Anterior uveitis
Leakage from retinal vessels
Ulceration (aphthous/genital)
Pustules after skin trauma (Pathergy test)
Scratching leaves lines (dermatographism)
Erythema nodosum
Thrombophlebitis
Remember posterior scleritis with POST SCLER
Proptosis
Ophthalmoplegia
Swelling of disc
Thickening of sclera (US/CT) & T sign (fluid in sub-Tenon’s space)
Subretinal exudates
Choroidal foLds
Exudative RD
Ring choroidal detachment
Remember causes of Choroidal neovascular membrane with HAMMAR
Histoplasmosis
ARMD
Multifocal Choroiditis
Myopia
Angiod
Rupture of the choroid
Remember the causes of trabecular pigmentation with PIGMENT
Pseudoexfoliation & Pigment dispersion syndrome
Iritis
Glaucoma (Post angle closure Glaucoma)
Melanosis of angle (oculodermal melanosis)
Endocrine (Diabetes & Addison’s Syndrome)
Naevus (Cogan-reese syndrome)
Trauma
Remember sterilization in ophthalmology with ABCDEFG
AUTOCLAVE
BOILING
CHEMICALS like Alcohol (Rectified spirit), Isopropyl alcohol/CIDEX: 2% Glutaraldehyde
DRY HEAT temperature of 150°C is used for 90 minutes
ETHYLENE OXIDE for sterilization of IOL etc.
FUMIGATION of operation theatre/ FORMALIN vapour
GAMMA-IRRADIATION: Gamma rays from Cobalt-60
Remember the ocular structures derived from neuroectoderm with MORE
Muscles of pupil
Optic Nerve
Retina (with RPE)
Epithelium of Iris
Epithelium of Cilliary Body
Remember the structures derived from surface ectoderm with S1L2E3
Skin of Eyelids and its derivatives viz. cilia, tarsal glands, conjunctival gland
Lens,
Lacrimal Gland,
Epithelium of Conjunctiva,
Epithelium of Cornea,
Epithelium of lacrimal passage
Remember Stromal dystrophies with Marilyn Monroe Always Gets Her Man in LA City.
Macular dystrophy
Mucopolysaccharide
Alcian blue
Granular Dystrophy
Hyaline material
Masson's Trichrome
Lattice Dystrophy
Amyloid
Congo Red
Remember the drugs causing cataract with ABCD
Amiodarone
Busulphan
Chlorpromazine
Dexamethasone
Remember the causes of Uniocular diplopia as ABCD
Astigmatism
Behavioral: psychogenic
Cataract
Dislocated lens
Remember microtropia with 3 A
Anisometropia
Angle small
Absent central field (Central suppression scotoma)
Remember the refractive indices of ocular media with 8303 (from anterior to posterior)
cornea 1.38
aq humour 1.33
lens 1.40
vit humour 1.33
Remember the types of colour blindness with TuB PaR DoG
TRITOANOPES = BLUE
PROTOANOPES = RED
DEUTROANOPES= GREEN
Remember the causes of lid retraction with 4 MP
4M= Myasthenia Gravis,Marcus Gunn jaw winkling syndrome,Myotonic causes like dystrophica myotonica.Metabolic cuses like uraemia,cirrhosis
4P=Perinauds syndrome,Parkinson's Disease/Progressive supranuclear palsy,Ptosis of other eye,Palsy (aberrant III crnaial nerve regeneration)
Remember content & veins draining the Cavernous Sinus with Rule of 3
3 Afferent veins: Sphenoparietal sinus (Vault veins), Supf Middle cerebral Vein (Brain), Ophthalmic vein (Orbit)
3 Efferent Veins: Superior petrosal sinus, Inferior Petrosal Sinus, Communicating vein to pterygoid plexus
3 Contents; Cranial Nerves (III,IV, V1,V2 & VI)
3 Areas Drain into it: Vault Bones, Brain (Cerebral Hemisphere), Orbit
3 Nerves: Motor(III,IV,VI),Sensory (V1,V2), Sympathetic
Remember the causes of pseudo tumor cerebri with " Idiopathic IDEA"
Idiopathic
Infections-Otitis media,mastoiditis,viral infections etc
Drugs-Steroid withdrawl,Vitamin A intoxication,Nalidixic acid,amidarone,cyclosporin,minocycline
Endocrine-obese,amennorrheic woman of child bearing age, Hypoparathyroidism
Anaemia
Remember the causes of downbeat nystagmus with DoWNBEAT
Degeneration, Demyelination or Drugs (Lithium)
Wernicke's Encephalopathy
Neoplasm or paraneoplastic cerebeller degeneration
Brainstem disease (Syringomyelia)
Encephalitis
Arnold-Chiari malformation
Trauma or Toxin
Remember characteristics of congenital nystagmus with CONGENITAL
Convergence & eye closure dampens
Oscillopsia absent
Null zone that is present,increases foveation time which results in increased acuity
Gaze poisition doesnot change the horizontal direction of nystagmus
Equal amplitude and frequency in each eye
Near acuity is good
Inversion of optokinetic response
Turning of head to acheive null point
Abolishes in sleep
Latent (occlusion) nystagmus occurs
Remember "DWARF" for decribing Nystagmus
Direction=plane of movement-horizontal,vertical
Waveform= Pendular or Jerky
Amplitude= fine or coarse
Rest=At primary position or gaze evolked
Frequency= How often the eye moves
Remember the characteristics of Perinaud's Dorsal Midbrain Syndrome with "CLUES"
Convergence retraction nystagmus
Lightnear Dissociation
Upgaze paralysis
Eyelid retraction
Skew deviation
Remember ocular features of acromegaly with ACROM
Angiod streaks
Chiasmal syndrome
Retinopathy
Optic atrophy,papilloedema
Muscle enlargement
Remember the systemic features of Marfan syndrome with MARFANS
Mitral prolapse
Aortic dissection
Regurgitant aortic valve
Fingers long (arachnodactyly)
Arm span>height
Nasal voice (high arched palate)
Sternal excavation
Remember the ocular features of Marfan’s syndrome with CLUMPS
Cupping (glaucoma)
Lattice
Upward lens subluxation
Myopia
Cornea Plana
Sclera blue
Remember angle structures with "I Can See Till Schwalbe's Line"
Iris root
Cilliary Body
Scleral spur
Trabecular Meshwork
Schwalbe's Line
Нүд чинь бүхнийг харуулна...
Thursday, August 26, 2010
Monday, August 23, 2010
Маш их хэрэгтэй хаягууд
http://eophtha.com/
Үүнд нүдний хэвийн хэмжээсүүд байна. Ёстой Тангад ном шиг цээжилнэ дээ. Лам нар өглөө болгон дүнгэнэтэл, навтартал уншина гэж үү?
Мөн маш сонирхолтой бас хэрэгтэй лекцүүд, санахад дөхөмтэй товчлолууд гэх мэт олон хэрэгтэй зүйлс байна.
http://www.eyeatlas.com/
Клиникийн хувьд их ач холбогдолтой юм. Нүдний сегмент болгоноор ангилсан байх юм.
http://www.redatlas.org/
http://dro.hs.columbia.edu/
Thursday, August 19, 2010
Tuesday, July 13, 2010
Types of cataract.
Cataract may be classified on the
-basis of stage of development
(e.g. intumescent, mature, hypermature),
-anatomical position of the
opacity (e.g. cortical, nuclear, subcapsular), or aetiology (diabetic, traumatic)
In clinical practice all 3 classifications tend to be used when describing a
cataract e.g. marked corticosteroid-induced posterior subcapsular opacities.
1. Intumescent:
When lens fibres degenerate to form a cataract there is breakdown of lens proteins into smaller molecules so that water passes into the lens which then swells. Such a lens may become so large that it causes severe shallowing of the anterior chamber and may close off the angle of the anterior chamber causing 'phakomorphic' glaucoma.
2. Mature:
A cataract is described as mature when the whole lens is opaque and no clear cortex is visible with a slit lamp. The mature cataract appears white and although the visual acuity is reduced to light perception, the ability to identify the direction of the light source is retained (normal projection to light). (LCW 4.4 p58) This important observation must always be recorded in these cases, as it gives some indication of the visual prognosis of a possible cataract extraction.
3. Hypermature:
In this stage the fens capsule in a mature cataract becomes permeable to liquefied lens matter which then leaks out into theaqueous. The cataract shrinks leaving a small brownish nucleus surrounded by a wrinkled capsule (Morgagnian cataract). Macrophages invade the aqueous
and the lens from the systemic circulation, engulf the lens matter and subsequently block the trabecular meshwork resulting in a rise in intraocular pressure ('phacolytic' glaucoma). Sometimes the cells sink to the bottom of the anterior chamber to produce the appearance of a hypopyon (sterile). The degree and speed of the pressure rise is also very variable. Lens induced
uveitis (phakoanaphylactic uveitis), due to the development of antibodies to lens protein after lens matter leaks into the anterior chamber, is to be distinguished from phacolytic glaucoma, although it may complicate it. After control of the intraocular pressure with osmotic diuretic agents e.g. diamox or mannitol and of the uveitis with corticostcroids, a lens extraction is performed. These lens induced conditions may also arise following lens capsule
rupture due to trauma or as a complication of surgery.
-basis of stage of development
(e.g. intumescent, mature, hypermature),
-anatomical position of the
opacity (e.g. cortical, nuclear, subcapsular), or aetiology (diabetic, traumatic)
In clinical practice all 3 classifications tend to be used when describing a
cataract e.g. marked corticosteroid-induced posterior subcapsular opacities.
1. Intumescent:
When lens fibres degenerate to form a cataract there is breakdown of lens proteins into smaller molecules so that water passes into the lens which then swells. Such a lens may become so large that it causes severe shallowing of the anterior chamber and may close off the angle of the anterior chamber causing 'phakomorphic' glaucoma.
2. Mature:
A cataract is described as mature when the whole lens is opaque and no clear cortex is visible with a slit lamp. The mature cataract appears white and although the visual acuity is reduced to light perception, the ability to identify the direction of the light source is retained (normal projection to light). (LCW 4.4 p58) This important observation must always be recorded in these cases, as it gives some indication of the visual prognosis of a possible cataract extraction.
3. Hypermature:
In this stage the fens capsule in a mature cataract becomes permeable to liquefied lens matter which then leaks out into theaqueous. The cataract shrinks leaving a small brownish nucleus surrounded by a wrinkled capsule (Morgagnian cataract). Macrophages invade the aqueous
and the lens from the systemic circulation, engulf the lens matter and subsequently block the trabecular meshwork resulting in a rise in intraocular pressure ('phacolytic' glaucoma). Sometimes the cells sink to the bottom of the anterior chamber to produce the appearance of a hypopyon (sterile). The degree and speed of the pressure rise is also very variable. Lens induced
uveitis (phakoanaphylactic uveitis), due to the development of antibodies to lens protein after lens matter leaks into the anterior chamber, is to be distinguished from phacolytic glaucoma, although it may complicate it. After control of the intraocular pressure with osmotic diuretic agents e.g. diamox or mannitol and of the uveitis with corticostcroids, a lens extraction is performed. These lens induced conditions may also arise following lens capsule
rupture due to trauma or as a complication of surgery.
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